A 13 year old boy is brought to ED by his parents with ankle pain after falling awkwardly on his ankle while skateboarding. He is unable to weight bear. His ankle x-ray is shown. What is the diagnosis?
Injuries to the traction epiphyses are avulsion injuries. Injuries to the pressure epiphyses at the end of long bones adjacent to the articular surface have been classified into five types— the Salter–Harris classification.
Note that Salter– Harris types I and V may not be apparent on the initial X- ray.
A concern specific to any epiphyseal injury is that premature fusion of a growth plate may result, with resultant limb shortening and deformity. The risk correlates to some extent with the mechanism of injury and amount of force involved. The different Salter– Harris fractures carry a different level of risk of long- term growth plate problems. The risk is low for types I and II (particularly if undisplaced), moderate for type III, and highest for types IV and V. Problems are usually averted if Salter– Harris type III and IV injuries are accurately reduced and held (e.g. by internal fixation). Type V fractures are notoriously difficult to diagnose and often complicated by premature fusion— fortunately, they are relatively rare.
This is an uncommon fracture, unique to adolescents because the distal tibial physis fuses from the medial to the lateral side. Beyond 16 years, the physis is closed and these injuries are not seen.
It occurs in the ankle of the young person (between a narrow window of approx. 12-16 years only) when a forced hyper-flexion and/or supination occurs at the ankle.
It is characterised by a Salter–Harris type III fracture through the anterolateral aspect of the distal tibial epiphysis with variable amounts of displacement.
Caution is required with Tillaux fractures because the Ottawa ankle rules applied to these injuries can miss them. This is because it is not uncommon for the patient to be able to weight bear and the defined bony points are non-tender. The tenderness is usually elicited at the anterior ankle joint line which Ottawa does not describe.
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Biochemistry | Normal Value |
---|---|
Sodium | 135 – 145 mmol/l |
Potassium | 3.0 – 4.5 mmol/l |
Urea | 2.5 – 7.5 mmol/l |
Glucose | 3.5 – 5.0 mmol/l |
Creatinine | 35 – 135 μmol/l |
Alanine Aminotransferase (ALT) | 5 – 35 U/l |
Gamma-glutamyl Transferase (GGT) | < 65 U/l |
Alkaline Phosphatase (ALP) | 30 – 135 U/l |
Aspartate Aminotransferase (AST) | < 40 U/l |
Total Protein | 60 – 80 g/l |
Albumin | 35 – 50 g/l |
Globulin | 2.4 – 3.5 g/dl |
Amylase | < 70 U/l |
Total Bilirubin | 3 – 17 μmol/l |
Calcium | 2.1 – 2.5 mmol/l |
Chloride | 95 – 105 mmol/l |
Phosphate | 0.8 – 1.4 mmol/l |
Haematology | Normal Value |
---|---|
Haemoglobin | 11.5 – 16.6 g/dl |
White Blood Cells | 4.0 – 11.0 x 109/l |
Platelets | 150 – 450 x 109/l |
MCV | 80 – 96 fl |
MCHC | 32 – 36 g/dl |
Neutrophils | 2.0 – 7.5 x 109/l |
Lymphocytes | 1.5 – 4.0 x 109/l |
Monocytes | 0.3 – 1.0 x 109/l |
Eosinophils | 0.1 – 0.5 x 109/l |
Basophils | < 0.2 x 109/l |
Reticulocytes | < 2% |
Haematocrit | 0.35 – 0.49 |
Red Cell Distribution Width | 11 – 15% |
Blood Gases | Normal Value |
---|---|
pH | 7.35 – 7.45 |
pO2 | 11 – 14 kPa |
pCO2 | 4.5 – 6.0 kPa |
Base Excess | -2 – +2 mmol/l |
Bicarbonate | 24 – 30 mmol/l |
Lactate | < 2 mmol/l |